Date of Award:

5-1-1975

Document Type:

Dissertation

Degree Name:

Doctor of Philosophy (PhD)

Department:

Biology

Department name when degree awarded

Bacteriology (Bacteriology and Public Health)

Committee Chair(s)

Rex S. Spendlove

Committee

Rex S. Spendlove

Committee

Paul B. Carter

Committee

Ross A. Smart

Committee

James L. Shupe

Committee

LeGrande C. Ellis

Abstract

The purposes of this investigation were to develop an improved method for the detection of anti-mycoplasmal antibody and the partial characterization of the effect of Mycoplasma arthritidis on lysosomal enzyme release from cells in culture. A new, rapid, sensitive and specific serologic test for the detection of anti-mycoplasmal antibody is described. This technique is based on the agglutination of acridine orange stained, whole mycoplasmas by homologous antiserum. The antiserum-aggregated, stained antigen is observed using the incident illumination fluorescence microscope. Titer of the antiserum is taken as the reciprocal of the highest antiserum dilution giving agglutination. Autoagglutination was a serious problem with certain mycoplasma species. However, the problem was overcome by using 10 percent fetal calf serum as the antiserum diluent. Optimal conditions for the fluorescent mycoplasma agglutination (FMA) test are: (1) Stain with acridine orange and harvest the antigen after 36-48 hours of incubation. (2) Incubate the stained cells with diluted specific antibody at 42 C for one hour. (3) Centrifuge the mixture at 700 X G for 10 minutes. (4) Place a drop of the antigenantibody mixture on a glass microscope slide and observe for agglutination. This test has been adapted to the microtiter system. The effect of M. arthritidis on lysosomal enzyme release from cells growing in vitro was assessed by monitoring the enzyme activity in the cell maintenance medium. It was found that the lysosomal enzyme, beta-glucuronidase, was released earlier and in larger amounts in the infected cell cultures. The cytoplasmic marker, lactate dehydrogenase (LDH), was not released in significantly larger amounts in infected cells. Variation in numbers of lysosomes was determined by vitally staining infected and non-infected cells with acridine orange. Infected cells always contained fewer lysosomes than did uninfected cells.

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